IL1B Blocking Peptide

Este producto es parte de IL1B - Interleukin-1 beta
IL1B Blocking Peptide
208€ (1 mg)

Por favor contáctenos para obtener información detallada sobre el precio y disponibilidad.

Name
IL1B Blocking Peptide
Category
Proteins and Peptides
Provider
Abbexa
Reference
abx061898
Tested Applications
BL

Description

IL1B Blocking Peptide is a blocking peptide which can be used to block the reactivity of the

Documentos del producto

Instrucciones
Data sheet
Descargar

Especificaciones del producto

Category
Proteins and Peptides
Immunogen Target
IL1B
Reactivity
Human, Mouse, Rat, Rabbit, Monkey
Purity
> 85%
Size 1
1 mg
Size 2
5 mg
Form
Lyophilized
Tested Applications
BL
Availability
Shipped within 7-12 working days.
Storage
Store at -20 °C for up to one year. Avoid repeated freeze/thaw cycles.
Dry Ice
No
Alias
IL-1F2, IL-1β, IL1β,IL-1 beta,IL1 beta,IL-1b,IL1b,IL1B
Background
Protein IL1B
Status
RUO
Note
THIS PRODUCT IS FOR RESEARCH USE ONLY. NOT FOR USE IN DIAGNOSTIC, THERAPEUTIC OR COSMETIC PROCEDURES. NOT FOR HUMAN OR ANIMAL CONSUMPTION.
Add blocking peptide to the diluted primary antibody in a molar ratio of 10:1 (peptide to antibody) and incubate the mixture at 4 °C overnight or at room temperature for 2 hours.

Background

IL1B is a major pro-inflammatory cytokine that belongs to the IL-1 family and plays a central role in inflammation, fever induction, and host defense. It is synthesized as an inactive precursor (pro-IL1B) in response to pathogen-associated molecular patterns (PAMPs) or damage-associated molecular patterns (DAMPs) and is cleaved into its active form by caspase-1 through inflammasome activation. IL1B binds to its receptor IL1R1, forming a signaling complex with IL1RAP, which activates downstream NF-κB, MAPK, and JNK pathways to drive the expression of cytokines, chemokines, and acute-phase proteins. IL1B is predominantly produced by activated monocytes, macrophages, and dendritic cells during infections, tissue damage, or inflammatory conditions. It plays a critical role in immune cell recruitment, pathogen clearance, and tissue repair, but excessive IL1B signaling leads to chronic inflammation, autoimmune disorders, and tumor progression. Dysregulation of IL1B is implicated in diseases such as rheumatoid arthritis, gout, and neuroinflammation. Knockout studies demonstrate impaired inflammatory responses, reduced cytokine production, and weakened host defense, highlighting IL1B's essential role in immune activation, inflammation, and disease pathogenesis.

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